trust me, youll feel a lot more focused and improved from taking stablon than any of the other smart drugs like paracetam or huperzine. stablon has a huge effect on neuroplasticity. stablon makes your brain process information faster and easier. its a real head clearer with no side effects. it feels good to lower excess serotonin, excess serotonin is so inhibitory, so stuplifying, makes you feel all weird, gets in the way. you know, MDMA doesnt owe its great effects just to serotonin release.. it actually lowers serotonin activity in the limbic system. Raised Serotonin being good is a myth, thats why people take SSRIs and feel like shit.

research stablon if youd like

http://www.tianeptine.com/
http://www.stablon.com/

Well Tone, I don't think trust comes into it!

I've been taking SSRI's for 15 years and have only witnessed improvements to my health.

From what I've read about Stabilon which works with an opposite mechanism to SSRI's, I admit that I am confused, since both work for different people.
Either we suffer from too much or too little serotonin in synapses, or maybe either an increase or decrease is beneficial.

A good experiment would be for SSRI users and Stabilon users to swap drugs and see what effect that has on each.

I read once somewhere that there is a theory that the serotonin antagonist LSD works by reducing serotonin initially. Then the psychedelic affect is caused by a rebounding inrush of serotonin. If that's true, why aren't the SSRIs psychedelics?

I'd prefer to get info from a place that doesn't sell the stuff. Piracetam has been researched for decades and seems to have no toxicity at all. Can you say the same for stablon? What have your experiences been with it and how long have you used it?

Difficult (as most/many studies are done from the company that produces the drug) but a careful search of pubmed may reveal some independent research.

I just took stablon and had heavy euphoria for 45 minutes followed by a clearing of my head rather than a crash. enjoy your SSRIs lol

How would you like to have a drug that does not use liver CYP nor kidneys to metabolize or clear? What if it was so safe that people took hundreds of tablets a day with no toxic effects? I think ill stick to using 4 tablets when i use it, i like mine to last.



----

The authors report a case of tianeptine abuse in a 30 year-old woman. After a medical prescription of the recommended dosage of 12.5 mg 3 times daily of oral tianeptine for a depressive illness, the patient spontaneously increased the dosage which after two months reached 150 tablets per day. No severe toxic effects were observed. As adverse effects, the patient, in the beginning of this high treatment period suffered from nausea, vomiting, abdominal pain, anorexia with weight loss, constipation. These side effects progressively disappeared. The biological tolerance was excellent, and hepatic parameters were not affected. The patient experienced and seek a psychostimulant effect. After seven months of such a therapy, she was hospitalized to undergo a withdrawal. The discontinuation of the tianeptine treatment occurs in four days. A withdrawal syndrome marked by myalgia, and cold feeling was transient, and alleviated by sedative phenothiazine (cyamemazine) and myorelaxant benzodiazepine (tetrazepam).

----

We report on a tianeptine dependence lasting for eighteen months in a 42 year old patient. The patient had a previous history of addiction to opiates, amineptine, cocaine and alcohol. He also had a family history of addiction to alcohol and opiates. Tianeptine was prescribed for a major depressive disorder. The patient alleged a "flash sensation" like with heroin since the very first doses with a physical and psychological well-being sensation, better psychomotor performances and transient mood elation. His addiction to tianeptine was immediate and heavy. The positive reinforcement faded away after one month and a total dependance took over, with physical and psychological withdrawal symptoms when doses were not renewed. After two months of treatment, the daily consumption of tianeptine was of 90 tablets. The patient was hospitalised to treat both the addiction to tianeptine and the ongoing major depressive disorder. He was taking 240 tablets daily. In the literature, reports of addictions to antidepressants are scarce and most of them involve agents with amphetamine-like properties, including amineptine and tranylcypromine. Other reports involving other antidepressant agents, including amitriptyline, fluoxetine and tianeptine remain exceptional. Addictions to antidepressants almost exclusively concern patients with a diagnosis of personality disorder and a previous history of drug or alcohol abuse and who are treated for a depressive disorder. Tianeptine, which is devoid of any psychostimulating effect in human, does not seem to have addictive properties apart from the reports of scarce cases.

http://www.biopsychiatry.com/tianeptine-addict.htm

Is tianeptine the same as stablon? It sounds addictive. There is always a price to pay for stimulants.

Yes it is the same. And no it's not addictive. Nor is it a stimulant.

Ssshhhhh! Or they'll stop prescribing them to us!

I don't know, I think I'll sit this out out for a while until a few years of reports come in. Maybe I'm missing out on a good thing but I just read some reports on modafinil over on that other site which shall remain nameless. Modafinil of course is not the same as this drug but some of the things said about it sound like what are being said about stablon. I know that proves nothing but stop to think that modafinil is approved over here by prescription, from what I've heard. It's been spoken of as a nootropic which I don't believe it is. The guy in europe who sells it said himself it was a stimulant. I heard from people who got hooked after a honeymoon period. Up to then, they thought it was the safest thing on the market. Now I'm hearing hype about ampekines (sp?). They are said to be wonderful much as stablon is said to be wonderful.

I'm not trying to warn anyone away from this new discovery. We need the early adopters much like we need canaries in the mine. It may be the best thing to come along since piracetam or even better. I'll just have to miss out for now. But keep those reports coming, I love to hear feedback about stuff like that. Anyone else been using stablon? Anybody ordered it, what does it cost?

"Guy in Europe" as da_sense??
Don't mix apples and pears. Modafinil is drug for narcolepsy. It can be used for other causes, but it's primarily stimulant, to keep one awake. Due to this it might "clear" mind for those who feel sleepy and such. But still it's a stimulant more similar to caffeine than Tianeptine.
Tianeptine on other hand is a antidepressive drug. But the research has shown some unique properties which i belive classify it as a nootropics as well. I've never used tianeptine so I can't comment on it's effects (i use deprenyl which has similar effects).
While you can get addicted to anything good, modafinil has much higher abuse potential, is you can feel it's effects right away and they can be strong if you take 500mg or more. Tianeptine is more subtle and not stimulatory and i don't think you can get addicted to tianeptine. At least i never heard someone got addicted to it.

BTW this got me thinking of trying tianeptine. But the cost of 3x tabs of tianeptine a day is much higher than 5 mg deprenyl daily...

You can take selegiline, not feel any smarter or any increase in focus at all, but be rest assured of antioxidant properties, or you can take stablon and actually feel results.

selegiline = deprenyl
i've been on 5 mg daily for the last 10 months, it really changed my life
best possible motivator, not so much for "feeling smarter" but can't beat it's motivation, and that's what i needed most

Is it safe to take deprenyl and tianeptine? On a long term basis..

For a healthy person yes.

I'm 26 and i started taking 5 mg deprenyl last june. I really changed my life. I found Deprenyl to be best for dopamine and motivation

[quote name='Promicarus' date='Jun 27, 11:51 AM' post='66667']
[quote name='Tone' post='61986' date='Mar 30, 08:41 PM']


having been diagnosed with "medication resistant depression" (gotta love the psyche terms--always seem to conveniently point the finger at the patient for having failed to improved under the respective Dr.'s care--COULDN'T be that he/she didn't have the slightest idea what they were doing...who me resentful?) Anyway..I've taken--while not EVERYTHING in the book, very nearly that..to no avail.
Happened across the tianeptine data online, and started looking into its mechanism of action--and low and behold, rather than "create a pool" of serotonin, which--the current U.S pharm theory dictates--it makes that serotonin more available to synapses which benefit from it-essentially, causes the synapses to SUCK it up, hungrily. And what happens when the body is using up anything...the body makes more!!!!!!
Turns out, tianeptine is a SSRP--selective serotonin reuptake PROMOTOR...and it would seem that, the more the synapses use-via tianep.--the more is naturally created! Add to this new data that much of the 'Pool' of serotonin the SSRI creates simply dissipates randomly into surrounding tissue-causing as-yet known effects--and it would seem as though the SSRI guys are going about it all the wrong way! Curious that it's banned in the U.S. huh?
Interestingly enough, it's also been demonstrated to promote neural growth in both the hippocampus and hypothalmus--those areas gradually eroded by excess stress--and that it's been tested with startling efficacy in those with chronic P.T.S.D. (who have cortisol related hypothalmus and hippocampus damage as a result of stress)--and it begins to become clear-is why it's been banned. After all--the FDA had NO JUSTIFICATION for having banned it, in the first place! No negative results to speak of, not even the odd, random negative incident! Thing is,it just may CURE people--and god forbid Pharm co.s got in the business of doing that! But that's another story...
No, I'm not denying your experience--If your SSRI has worked for you, great. Problem with SSRIs? Your brain eventually "realizes" that there's far too much serotonin around, much more than is being used...so it DOWN REGULATES SEROTONIN PRODUCTION!--AND THEN WHAT ARE YOU STUCK WITH?..THE NEED TO TAKE MORE AND MORE, JUST TO GET THE SAME EFFECT...AND WORSE, THE INABILITY TO QUIT,AS YOUR BODY'S ALREADY--STOPPED PRODUCING THE SEROTONIN IT SHOULD BE--AND EVEN WAS IN THE FIRST PLACE!!! YOU'RE HOOKED, BABY, THAT'S WHAT. A Pharm corp. junkie bit*h. ASK THOSE WHO'VE TAKEN PAXIL, ASK ABOUT PROZAC DOSAGE ESCALATION....THEN ASK YOURSELF AGAIN WHETHER YOU'RE really BETTER OFF--- IN THE....LONG RUN.
Tianep has even been demonstrated as having a positive effect on the HPA axis--that's the gland/brain conjunction which is responsible for our "losing our sh*t" in stressfull situations--no not through sedation, by any means. In fact, it's been demonstrated to have both some stimulating and some anxiolytic properties-but no real "sedation"to speak of (and believe me, I know sedation)

Maybe an excess of serotonin leeching indiscriminately into your brain tissue is what you need--there is a calming affect associated with it, after all. But don't kid yourself that it's making any difference where the efficiency of effectiveness of natural, optimally effective neural transmissions are concerned.
Right on about the neuroplasticity,too--if you don't believe, do the research yourself--but don't scoff--scoffing is for armchair cynics. By the way--it's the first thing--of the probably 20 I've tried-ove the last 15 years of continuous searching, that actually WORKS--I'd long ago given up hope, of having a relatively normal life, as a matter of fact, must less enjoying it in the least. So don't--give up-- until you've tried tianept.

Truth.

Serotonin is not a serotoninc "antagonist"--it's serotonin-related receptor site agonist --meaning it "opens them all up"--in hippie lingo, so to speak. As far as LSD actually promoting the production of serotonin, I'm not sure.
But it does enable the serotonin that's there to have completely free reign.

Obviously, your concern regarding the longstanding record of Stablon (Tianeptine) is valid. However, it has been in production and has been prescribed regularly throughout Europe for approx. 25 years, now--with no accompanying reports of adverse reactions. That's certainly more than can be said for the majortiy of most recent releases in the U.S. market. Obviously, again, one must weight his subjective need with the degree of risk with which he is comfortable assuming in attmept to meet those needs. Nothing is absolutelywithout risk. I'm sorry to inform you that you may be the first, one and only, individual ever to suffer painful death (or for that matter,any slight inconvenience) from having taken Stablon--but yes, you make a good point: That's always a possibility. It certainly is nice when others perform independent research before asking pointed and derrogatory questions--but I suppose that's not nearly as fun,is it?

Got a maybe dumb question about tianeptine usage. I'm thinking of taking once per day (instead of the standard x3 dose) at night before sleep. Is there any point in doing so? Will it work?

I am not depressed at the moment but I had some issues in the past - I want to take it as a smart drug, for it's neural growth properties and because when I was on Tianeptine I handled stress better and I could sleep more easily. So I want to give a subtle long term solution with this scenario.

Anybody?

I may have missed something here. Is stablon illegal in the USA? I didn't realise it had been used that long in europe. I still would like to hear more research on it.

So? if you take a dopaminergic your dopamine receptors downregulate. if you take an opioid, your mu downregulates. if you take a noradrenalergic your noradrenaline receptors downregulate. also, i get tolerance to stablon fast, granted it also has long term benefits.

comparing stablon to SSRIs is literally like comparing SSRIs to cocaine or percocet. stablon causes pleasure ok, it makes you feel well and stimulated. SSRIs do not, otherwise youd see them being sold on the street and also patients saying they love it so much.

Order it, its not illegal, why would it be illegal?? its just not a drug thats here thats all.

Bump!?

Yea. why not? one is a lot lot more than none. you could take one and still get some benefit im sure. stablon is very good for your brain =)

Its one of the safest drugs for depression I've come across.

Thanx Tone and LifeMirage!

Does anyone know anything about it's effects on dementia patients?

How does it effect schizophrenics?

Maybe these are helpful:

[The study of the efficacy of fluoxetine versus tianeptine in the treatment of elderly depressed patients followed in general practice]

Guelfi JD, Bouhassira M, Bonett-Perrin E, Lancrenon S.

Universite Paris XI, Hopital Paul-Brousse, Villejuif.

Depression in the elderly appears to be frequently poorly understood and underdiagnosed. The frequency of depressed illness in subjects over the age of 65 years is an important problem of health care. General practitioners are often in the first line of care. The objective of this trial was to compare the efficacy and safety of fluoxetine 20 mg/day to tianeptine 37.5 mg/day in elderly patients suffering from major depressive episode defined according to DSM III-R criteria, Newcastle Depression Scale < or = -20, without associated dementia Mini Mental Status (MMSE), treated by general practitioners in ten different french regions during 3 months. 237 patients were randomised, each patient had to be treated for 12 weeks and was reviewed 5 times during the trial. Patients had signed an informed consent. This trial was in favor of the superiority of fluoxetine compared to tianeptine. The main criterium, defined as the difference of the Montgomery and Asberg Depression Rating scale (MADRS) score between day 0 and day 84, was significantly different in favour of the fluoxetine group (intent to treat and per protocol analysis) (p = 0.019). The success rate at the end of the treatment (MADRS < or = 10) was significantly higher in the fluoxetine group (fluoxetine group: 48.4% vs tianeptine group: 28.1%) (p = 0.005). These results were confirmed by analysis of the other assessment criteria Geriatric Depression Scale (GDS) and Clinical Global Impression (CGI). During the study, the safety of the two treatments was comparable.

PMID: 10434153




Prevention of stress-induced morphological and cognitive consequences.
McEwen BS, Conrad CD, Kuroda Y, Frankfurt M, Magarinos AM, McKittrick C.

Laboratory of Neuroendocrinology, Rockefeller University, New York, NY 10021, USA.

Atrophy and dysfunction of the human hippocampus is a feature of aging in some individuals, and this dysfunction predicts later dementia. There is reason to believe that adrenal glucocorticoids may contribute to these changes, since the elevations of glucocorticoids in Cushing's syndrome and during normal aging are associated with atrophy of the entire hippocampal formation in humans and are linked to deficits in short-term verbal memory. We have developed a model of stress-induced atrophy of the hippocampus of rats at the cellular level, and we have been investigating underlying mechanisms in search of agents that will block the atrophy. Repeated restraint stress in rats for 3 weeks causes changes in the hippocampal formation that include suppression of 5-HT1A receptor binding and atrophy of dendrites of CA3 pyramidal neurons, as well as impairment of initial learning of a radial arm maze task. Because serotonin is released by stressors and may play a role in the actions of stress on nerve cells, we investigated the actions of agents that facilitate or inhibit serotonin reuptake. Tianeptine is known to enhance serotonin uptake, and we compared it with fluoxetine, an inhibitor of 5-HT reuptake, as well as with desipramine. Tianeptine treatment (10 mg/kg/day) prevented the stress-induced atrophy of dendrites of CA3 pycamidal neurons, whereas neither fluoxetine (10 mg/kg/day) nor desipramine (10 mg/kg/day) had any effect. Tianeptine treatment also prevented the stress-induced impairment of radial maze learning. Because corticosterone- and stress-induced atrophy of CA3 dendrites is also blocked by phenytoin, an inhibitor of excitatory amino acid release and actions, these results suggest that serotonin released by stress or corticosterone may interact pre- or post-synaptically with glutamate released by stress or corticosterone, and that the final common path may involve interactive effects between serotonin and glutamate receptors on the dendrites of CA3 neurons innervated by mossy fibers from the dentate gyrus. We discuss the implications of these findings for treating cognitive impairments and the risk for dementia in the elderly.

PMID: 9405958

Control of ACTH secretion by excitatory amino acids: functional significance and clinical implications.
Jezova D.

Laboratory of Pharmacological Neuroendocrinology, Institute of Experimental Endocrinology, Slovak Academy of Sciences, Vlarska 3, Bratislava 83306, Slovakia. ueenjezo@savba.sk

The involvement of excitatory amino acids in the control of ACTH release is well established. Activation of ionotropic glutamate receptors has a stimulatory effect on ACTH release, while the role of metabotropic receptors is not yet understood in detail. Glutamatergic regulation of ACTH release has a clear significance for the stress response and neuroendocrine functions during development. A dysregulation of the hypothalamic-pituitary-adrenocortical (HPA) axis has been reported in several psychiatric and neurological disorders. So far, only fractional indices on the clinical importance of the interaction between glutamate and ACTH secretion have been obtained in both preclinical and clinical studies. Some antidepressant drugs, such as tianeptine, which were found to modulate ACTH release, appear to interfere with brain glutamatergic system. Changes in ACTH and cortisol release may be of importance for mood stabilizing effects of antiepileptic drugs modulating glutamate release, such as lamotrigine. Brain glutamate and HPA axis interaction seems to be of importance in alcohol and drug abuse. Little information is available on ACTH release in response to glutamate-modulating drugs used in the treatment of schizophrenia and Alzheimer disease. Nevertheless, pharmacological interventions influencing interaction between glutamate and the HPA axis are promising treatment possibilities in psychiatry and neurology.

PMID: 16388118



Plasticity at hippocampal to prefrontal cortex synapses is impaired by loss of dopamine and stress: importance for psychiatric diseases.
Jay TM, Rocher C, Hotte M, Naudon L, Gurden H, Spedding M.

INSERM E 0117, Physiopathologie des Maladies Psychiatriques, Centre Paul Broca, 2ter rue d'Alesia, 75014 Paris, France. jay@broca.inserm.fr

The direct hippocampal to prefrontal cortex pathway and its changes in synaptic plasticity is a useful framework for investigating the functional operations of hippocampal-prefrontal cortex communication in cognitive functions. Synapses on this pathway are modifiable and synaptic strength can be turned up or down depending on specific patterns of activity in the pathway. The objective of this review will be to summarize the different studies carried out on this topic including very recent data and to underline the importance of animal models for the development of new and effective medications in psychiatric diseases. We have shown that long-term potentiation (LTP) of hippocampal-prefrontal synapses is driven by the level of mesocortical dopaminergic (DA) activity and more recently that stress is also an environmental determinant of LTP at these cortical synapses. Stimulation of the ventral tegmental area at a frequency known to evoke DA overflow in the prefrontal cortex produces a long-lasting enhancement of the magnitude of hippocampal-prefrontal cortex LTP whereas a depletion of cortical DA levels generates a dramatic decrease in this LTP. Moreover, hippocampal stimulation induces a transient but significant increase in DA release in the prefrontal cortex and an optimal level of D1 receptor activation is essential for LTP expression. We recently investigated the impact of stress on hippocampal-prefrontal LTP and demonstrated that exposure to an acute stress causes a remarkable and long-lasting inhibition of LTP. Furthermore, we demonstrated that tianeptine, an antidepressant which has a unique mode of action, and clozapine an atypical antipsychotic when administered at doses normally used in human testing are able to reverse the impairment in LTP. Stressful life events have a substantial causal association with psychiatric disorders like schizophrenia and depression and recent imaging studies have shown an important role of the limbic-cortical circuit in the pathophysiology of these illnesses. Therefore, we proposed that agents capable of reversing the impairment of plasticity at hippocampal to prefrontal cortex synapses have the potential of becoming new therapeutic classes of antidepressant or antipsychotic drugs.


PMID: 15325962

Thanks kLee. That was very helpful.

I just found this place yesterday and have recently been introduced to Stablon, though I haven't tried it yet. But it appears to be exactly what I need. I basically suffer from PTSD, as a result of an abusive parent. I truely believe there has been severe damage done to my hippocampus and hypothalamus, as a result. Over the past month I've been using CES (cranial electro-stimulation) to pretty good results. There is evidence that CES works to stimulate and heal parts of the limbic system.

Anyway, I'm really excited to add Stablon into the mix. I'm weary though of meds, but this one appears much different from your standard SSRI's. I've been on Paxil before and I don't want to go through that hell again.

Thanks for the invaluable info on Stablon and I'll let you know my progress as it comes.

-Lummar

Lummar, any progress? What happened?

Will Stablon have any effect taking it the first time or it has to be used for a long time before noticing any effects? Thanks!

Hi! :-) I've been on Stablon since March 2005 for major depression and panic disorder at a dosage of 12.5mg QID (4x/day) and didn't notice any effects until day 12 of treatment (12.5mg TID). I felt a sudden onset of what seemed to be adrenaline (although that's probably not what it was) and it lasted for a few days along with feeling jittery but it didn't give me any panic attacks (I have panic disorder, so I'm prone to them). A reduction in appetite was noted. I adjusted the dosage during this time of "activation" and with time I felt my mood improving and anxiety and other general tension decreasing - all without any sedation whatsoever. The jitteriness and "adrenaline-ish" feelings subsided after a few days. SSRIs, on the other hand, produced far worse side effects (for me anyway). I haven't been able to tolerate any antidepressant as well as I have been able to tolerate Stablon...and this one lacks cardiotoxicity since it doesn't affect the sodium channels in the heart.

It took about 4 to 6 weeks to feel the full antidepressant effect and with time its anxiolytic effect becomes more fully pronounced. I do best on 4 tablets a day, rather than 3. I felt three tablets per day to be subtherapeutic, but everyone reacts to medications differently.

Do you have asthma? I do and it really made a difference there. Stablon improves the clinical picture of asthma by removing free serotonin from the bloodstream. Read this website on wikipedia to find out more about it: http://en.wikipedia.org/wiki/Tianeptine

If I take two Stablon at once, I can feel a swift mood brightening effect which is pleasurable and I feel more social and better able to concentrate. Rarely, I will experience an "Ecstasy"-like effect where I feel so comfortable and want to touch or hug something... this never happened with any other antidepressants I've tried. It's only happened 5 times or less in the entire time I've been on Stablon, but I loved that. I have never taken MDMA though, but when I described this unique effect to friends with knowledge of MDMA, they said this sounds like a very mild "Ecstasy"-like effect.

I've never experienced any withdrawal symptoms from it. When I did reduce the dosage (all the way down to 1 tablet per day, albeit gradually over a 2 to 3 week period), I just felt anxiety and depression returning, but not in any rebound sort of fashion.

I'm also on diazepam (Valium) and Stablon seems to make it much easier to taper downward my dosage. Stablon has been shown to block panic attacks as effectively as Paxil.

The reason you must take it at least 3 times per day is because it has a short half-life.

I am glad I found Stablon. I can also get Survector (amineptine) and you are welcome to ask me about how to get it legally, because it is legal in many countries and it is still available where I live, although it is just remaining stock. For example, it is not scheduled in the US or the UK.
I know a lot of people have asked me about Survector and Stablon in other forums on the internet. I would recommend it for non-neurotic depression and recommend Stablon for any type of depression, especially depression with neurotic features as it is anxiolytic too.
I would especially recommend it for anyone who wants to preserve their sex drive, because as is well-known, SSRIs destroy the libidos of many and have even caused what is known as PSSD (Post-SSRI Sexual Dysfunction) which persists for months and even years after discontinuing the SSRI.

Recent research has shown that it does increase dopamine in the nucleus accumbens and cortex and this may play a role in its antidepressant effect and why this drug has no sexual side effects. If anything, it may increase libido, but I haven't seen any studies. Oops, there is a study on that website I listed earlier in my post... a study done in Egypt where it helped men with erectile dysfunction and associated depression.

I just think, in conclusion, that the full effect of Stablon will make itself known in the long term. I know that for myself I have noticed more benefits in the long term (i.e. months). It has helped my depression, anxiety and asthma and I would never give it up for anything else.

Hi Survector,

A very informative post there!

In particular, you point out that it is for everyone to find their 'best' medicine.

I would love to try stabilon since good reviews abound, but can't for the life of me understand why it should still be effective when it works in the opposite way to most other antidepressants.

[/quote]

I can also get Survector (amineptine) and you are welcome to ask me about how to get it legally, because it is legal in many countries and it is still available where I live, although it is just remaining stock. For example, it is not scheduled in the US or the UK.

[/quote]


I would be interested in knowing how to purchase Survector (amineptine) legally.


Let me know.

Thanks

Me too - ive tried some "research grade" amineptine & would like to try the original survector product before crossing it off my list..

I am a very big supporter of Stablon...and I believe no other drug in the antidepressent catagory is so good as stablon...it really changed my life .....when I was having depression.....I 100% recommand it.. though on your own risk and with appropriate causion...different drugs effect differently with each other...so...

My doctor just prescribed me effexor xr . I took one pill and researched this drug and no way I'm taking that poison. I am very interested in Stablon and little interested in Survector. Where is the best place to buy these?

Jimiam

PhilMcCock,

Google : Buy Stablon....

Most people buy it from "AuraPharm" named website....but it takes 4 weeks to arrive....though it is comparatively cheap.

I trust this has been some time since a last post. I would like also access to SURVECTOR if possible. Thank you an advance.

See supplier list for a safe secure place to order.

Hello all,

I have been battling depression for years and have found Stablon to be the first drug that has actually helped me with no side effects. The problem is that I'm in the US and for reasons I can't go into here, I need a legal prescription. I would be willing to go to France if I had to, but does anybody know of the closest place to the US where I can see an MD to get a prescription? Is it possible, for example, to get it in Bermuda or Canada? If anybody knows an MD I could contact, it would really help.

With thanks in advance,

Seth

Have it sent to a PO box.

Does anyone know a present source for amineptine? I have come across one that asks a payment with Western Union but I'm not sure about the quality of the source. Amineptine and Emsam sound like best solutions for my depression. I find it ironic that neither of the medicines can be bought from Europe at the moment, even with prescription.

-Cx